VO₂ Max and Aging: What’s Normal vs. What’s Preventable

You’re supposed to lose some capacity. But not this much—and not this fast.

The curve everyone talks about—but almost no one questions.

It’s the same in study after study:

VO₂ Max peaks in your 20s, then slides down the slope—about 10% per decade after 30.

By the time you hit your 70s, the chart suggests you’ve lost nearly half your aerobic capacity.

The implication is quiet but clear: That’s just aging.

Except… it’s mostly not.

The Typical Decline

Longitudinal studies show an average drop of ~10% per decade in adults [1]:

Age Average Men
(ml/kg/min)
Average Women
(ml/kg/min)
20–29 45 38
30–39 41 35
40–49 37 32
50–59 33 29
60–69 29 26
70+ 25 22

Look at those numbers in functional terms:

  • 45 ml/kg/min = running a 5K without much trouble

  • 25 ml/kg/min = struggling up a few flights of stairs

That decline has consequences—far beyond race times.

Why VO₂ Max Falls

Some change is biological:

  • Maximum heart rate drops ~5–10 bpm per decade

  • Mitochondria lose efficiency in energy production

  • Capillary density can decrease

  • Muscle mass and strength shrink with disuse

In women, the slope can steepen after menopause—dropping 12–15% per decade if sedentary—because estrogen loss accelerates muscle and capillary decline. The same training that slows decline in men produces similar protection in women [4,5].

Biological vs. Behavioral

Think of VO₂ Max decline as two layers:

  1. Biological aging — inevitable physiological change

  2. Behavioral aging — the signal you send by what you do (or don’t do)

For most adults, the behavioral component dominates.

Reviews estimate that 50–70% of VO₂ Max decline is preventable through consistent training and physical activity [6].

The biology sets the slope; behavior decides how steep it gets.

How Training Changes the Curve

Aerobic training—especially a mix of Zone 2 base and Zone 4 peak work—can:

  • Preserve mitochondrial density

  • Maintain higher stroke volume

  • Slow heart rate decline

  • Preserve lean muscle and capillaries

Even in adults over 60, 8–12 weeks of structured Zone 2 and Zone 4 training can boost VO₂ Max by 5–10%, according to multiple meta-analyses [7].

Capacity and Independence

Research shows:

  • <20 ml/kg/min → high risk of losing the ability to live independently

  • <15 ml/kg/min → daily activities become exhausting

These thresholds are age- and sex-adjusted; for example, independence cutoffs may be closer to ~20 ml/kg/min for men and ~18 for women in their 70s [3].

Above these cutoffs, recovery from surgery or illness is faster, fall risk is lower, and hospital stays are shorter.

What “Preserved Capacity” Looks Like

Active septuagenarians with VO₂ Max in the mid-30s:

  • Walk briskly without breathlessness

  • Carry groceries upstairs

  • Hike for hours

  • Bounce back from minor illness or injury in days—not weeks

These aren’t genetic outliers—they’re people who kept training.

How DexaFit Tracks It

Repeat testing every 6–9 months lets you:

  • See your current percentile for age and sex

  • Measure change against your own baseline (not just population averages)

  • Identify whether decline is tracking biological pace—or accelerating due to lifestyle

With those data points, you can adjust training before the loss becomes obvious in daily life.

Final Thought

Yes—VO₂ Max declines with age. But the steepness of the slope is largely yours to choose.

Most people lose capacity twice as fast as they need to. They call it aging. It’s not—it’s underuse.

Every preserved MET (3.5 ml/kg/min) cuts mortality risk by roughly 13% [8].

When you hold on to capacity, you’re not just staying fit—you’re quietly shifting the odds in your favor for every year that follows.

Keep sending the signal. Climb the hills. Sprint the straights. Build the base.

Not to stop the clock—but to make sure, when the decades pass, you’re still strong enough to use the time you’ve earned.

Age will lower your ceiling. Training decides how high it still is when you need it most.

References

  1. Fleg JL, et al. Circulation. 2005;112(5):674–682.

  2. Trappe S, et al. J Appl Physiol. 1996;80(2):743–750.

  3. Arena R, et al. Am J Cardiol. 2007;99(3):377–382.

  4. Fitzgerald GK, et al. Menopause. 2020;27(3):1–9.

  5. American College of Sports Medicine. ACSM’s Guidelines for Exercise Testing and Prescription. 11th ed. 2021.

  6. Hawkins SA, Wiswell RA. Rate and mechanism of maximal oxygen consumption decline with aging. Sports Med. 2003;33(12):877–888.

  7. Huang G, et al. Exercise training and cardiorespiratory fitness in older adults: meta-analysis. Ageing Res Rev. 2016;28:49–61.

  8. Kodama S, et al. Cardiorespiratory fitness as a quantitative predictor of all-cause mortality and cardiovascular events. JAMA. 2009;301(19):2024–2035.